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FAK-paxillin activation for fibroblast migration Growth hormone receptor upregulation on tendons VEGFR2-Akt-eNOS pathway for blood vessel formation Nitric oxide modulation reducing inflammation Erg-1 transcription factor inhibition Angiogenesis, the formation of new blood vessels, represents another critical mechanism
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In models of NOS inhibitor-induced hypertension, BPC-157 attenuated blood pressure changes, suggesting interaction with the NO pathway rather than simple NO donation
